It is theorized that these multiple “causes” interact with each other in subtle and complex ways, and would thus explain the wide range of differing outcomes and behaviors in each individual. Damasio and Mauer have also proposed that “the syndrome results from dysfunction in a system of bilateral neural structures that includes the ring of mesolimbic cortex located in the mesial frontal and temporal lobes, the neostriatum, and the anterior and medial nuclear groups of the thalamus.” (Noteworthy, is that much emphasis is put on the medial temporal lobe). Schmidt & H. Given the neurodevelopmental nature of the disorder it is unsurprising that onset of symptoms is usually observed early during postnatal development. Is it really just a reactivated herpes virus or is it actually some other kind of lethal virus that can be passed from person to person and can become chromsomally integrated? Researchers discovered that 83% of kids with autism had an antibody from the measles virus that was directed against a protein. 1 HSV: herpes simplex 1 causes ulcers in the mouth.
They cause immune abnormalities of T lymphocytes (one type of white blood cell); they induce the production of autoantibodies; they involve hormonal factors; and they generally show a gender preference. Herbs with anti-viral properties are included in each formula, and specific “guide herbs” are included to target the precise area of the body where lesions occur. Facial recognition is also severely impaired, although not to the point of being face-blind, as autistic people use the wrong part of the brain to process faces (essentially processing faces in the same place as objects), and, unlike people without autism, they process faces as individual pieces and at the same speed as everything else. Animal models in which early viral infection results in behavioral changes later in life include the influenza virus model in pregnant mice and the Borna disease virus model in newborn Lewis rats. Basically, the doctors had left us with no options but to “research it ourselves”. Likewise, autism shows a greater concordance rate in monozygotic twins than in the normal population.7 Autism is also four to five times more prevalent in boys than in girls � a gender factor which is also seen in systemic lupus erythematosus (SLE), Grave’s disease, and ankylosing spondylintis (though this is more common with women than men). There are several identified susceptibility genes that raise the risk of autism.
As reported in The Lancet Infectious Diseases, Ana de Filippis (Oswaldo Cruz Institute in Rio de Janeiro, Brazil) and team studied two women (aged 27 and 35) who had displayed symptoms of Zika infection including rashes, fever and muscle pain during the first trimester of pregnancy. UC Davis 17 Oct 2002. Since this scared several moms on the board, we decided to take a look at that and some other papers that infer such an association. Immunological studies of autistic patients have revealed certain features that are also found in patients with other autoimmune diseases. This is consistent with the idea of an immune dysfunction / dysregulation. One received flu mist, despite being too young to receive it. Studies have also been done comparing the maternal antibodies of mothers with their autistic children.
What was once a relatively rare disorder is now twenty times more likely to occur. I was infected with that as an adult. These disturbances in energy metabolism were not due to genetic mutations, which is often seen in mitochondrial problems, but a condition the children studied acquired in utero or after birth. Encephalitis, which is defined as inflammation of the brain, is medical diagnosis code G04.90 in the International Classification of Disease, 10th revision; however, children with an ASD diagnosis are not generally assessed for a possible medical diagnosis of encephalitis. Environmental and genetic factors do contribute to the development of autism, but most children with autism have normal health, with no medical reason for the autistic symptoms. Serotonin has been shown to modulate some immune responses and hyperserotonemia may explain some of the abnormal cellular immune responses seen in autism. Digestion is impaired and slowed if the vagus nerve is not signalling optimally.
The program’s narrator stated there had been “a puzzling jump in the number of children being diagnosed with autism.” However, the number being diagnosed may reflect increased reporting of cases rather than an increase in actual incidence. Viruses tend to activate when exposed to excessive levels of sugar, carbohydrates. In the 1950s and 1960s it was still generally believed that this parental behavior caused autism. According to TACA (Talk About Curing Autism), infections that are implicated in the medical literature as causing or triggering cases of autism include: Herpes, Rubella, Mycoplasma Pneumoniae, Shingella, Borna, Malaria, Blastocystics, Varicella, Cytomegalo Virus (CMV), Syphilis, Toxplasmosis, Neurocysticercosis, and Clostridium. The line was crossed when attenuation ended and gene vaccines were born.